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In a rat model, our previous research on early NHPN-1010 treatment for blast-induced tinnitusspecifically targeting cochlear synaptopathydemonstrated that (1) blast overpressure caused a permanent loss of cochlear nerve ribbon synapses (RSs), and (2) administration of NHPN-1010 within 3 days of blast trauma, compared to placebo, significantly rescued RSs, as evidenced by ribbon counts in cochlear histological analyses and corresponding functional changes in suprathreshold wave-I amplitudes of the auditory brainstem response (ABR) 34

Peptide-functionalized LNPs, guided by exenatide-derived ligands, have additionally achieved targeted mRNA delivery across the blood-brain barrier, establishing a framework for neuroendocrine applications of GLP-1R-targeted gene therapy [116, 427]