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Thus, the accumulation of free radicals in the body could trigger an array of non-infectious diseases
3a) Consistent with a deleterious role for free iron in ASCVD, ApoE-/- mice exposed to chronic iron overload displayed exacerbated atherosclerosis likely secondary to VEC damage or death, which was attenuated by dietary iron restriction or iron chelation 62,63
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Oxidative stress and exposure to neurotoxins may also induce conformational changes in alpha-synuclein, promoting its aggregation into toxic forms
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