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Because animal growth occurs with the active production of free radicals and ATP consumption, robust antioxidative and energy transformation systems serve as the prerequisite for rapid protein gain and optimal health [25, 26]

Inhibition of GCH1 reduced BH4 and resulted in enhanced erastin-induced iron accumulation and lipid peroxidation, thereby enhancing ferroptosis sensitivity, while BH4 supplementation suppressed ferroptosis-related changes caused by GCH1 downregulation